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Potent selective inhibitor of protein kinase G I alpha, I ß and especially of type II. Activates retinal cGMP channels and thus discriminates between kinase and channel effects. Absolutely resistant against mammalian cyclic nucleotide-dependent phosphodiesterases, no metabolic side effects. Significantly more lipophilic and membrane permeant compared to Rp-cGMPS (Prod. No. BLG-G016)or Rp-8-Br-cGMPS (Prod. No. BLG-B005).
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