IL-22 receptor antagonist (Y51A) , pegylated (human), (recombinant)

I-TIF

Recombiant human IL-22 Y51A mutant is biologically active, capable of full inhibition of mouse IL-22 induced STAT3 phosphorylation in HepG cells. Its inhibitory acrtivity in vitro is ~ 10-20% compared to the non-pegylated mIL22 (Y51A) mutant.

< 0.1 ng/µg of protein (<1EU/µg)

Lyophilized.

36.0 kDa (monomer)

> 98.0% as determined by Gel filtration and SDS-PAGE gel.

Produced in E. coli.

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