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Cytochalasin E

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Inhibits angiogenesis

An aspergillus derived fungal metabolite. Inhibits angiogenesis and tumor growth. An actin microfilament depolymerizing agent.

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Appearance	White solid.
CAS	36011-19-5
Formula	C₂₈H₃₃NO₇
MW	495.6
Purity	≥98% (HPLC, TLC)
Solubility	Soluble in DMSO (50mg/ml) or ethyl acetate (5mg/ml)
Source	Isolated from Aspergillus clavatus

Use/Stability	As indicated on product label or CoA when stored as recommended. Store, as supplied, at -20°C for up to 1 year. Store solutions at -20°C for up to 3 months.
Handling	Protect from light.
Long Term Storage	-20°C
Shipping	Ambient Temperature

Regulatory Status	RUO – Research Use Only

Depolymerization of actin filament by cytochalasin E induces interleukin-8 production and up-regulates CD54 in the HeLa epithelial cell line: N. Ikewai et al.; Microbiol. Immunol. 47, 775 (2003), Abstract
Cytochalasin E, an epoxide containing Aspergillus-derived fungal metabolite, inhibits angiogenesis and tumor growth: T. Udagawa et al.; J. Pharmacol. Exp. Ther. 294, 421 (2000), Abstract
Actin depolymerization and polymerization are required during apoptosis in endothelial cells: N. Suarez-Huerta et al.; J. Cell. Physiol. 184, 239 (2000), Abstract
Cytochalasin-E-Resistant Mutants of Coprinus cinereus: Isolation and Genetic, Biochemical, and Cytological Analyses: M. Tsukamoto et al.; Fungal Genet. Biol. 20, 52 (1996), Abstract
Inhibition of actin polymerization in blood platelets by cytochalasins: J.E. Fox & D.R. Phillips; Nature 292, 650 (1981), Abstract

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